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Sagot :
Answer:
The correct answer is ''C. Renin-angiotensin system activation causes decreased glomerular filtration rate and fluid retention, which results in increased preload.''
Explanation:
In states of low cardiac output, the renin-angiotensin-aldosterone pathway is activated as a compensatory mechanism, whose objective is to fundamentally modify the peripheral circulation, which operates in conjunction with the activation of the medullary neuro-adrenal system, to maintain blood pressure. These two mechanisms, which are totally different, are closely linked; thus, stimulation of beta-1 adrenoreceptors in the juxtaglomerular apparatus, as a consequence of a high adrenergic output, causes renin output in heart failure. On the other hand, the activation of baroreceptors in the renal vascular bed caused by a reduction in renal blood flow, is also responsible for the release of renin, it occurs in patients with severe heart failure managed with diuretics and salt restriction.
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