Answered

Find the best answers to your questions at Westonci.ca, where experts and enthusiasts provide accurate, reliable information. Discover in-depth answers to your questions from a wide network of professionals on our user-friendly Q&A platform. Discover in-depth answers to your questions from a wide network of professionals on our user-friendly Q&A platform.

A cell that utilizes NHEJ to repair a double-stranded DNA break which occurred in the open reading frame of a gene is susceptible to indels resulting in a frame shift mutation and ultimately a non-functional protein. False True

Sagot :

luisvaschetto

Answer:

True

Explanation:

The non-homologous end-joining (NHEJ) pathway is a DNA repair pathway used to repair double-strand breaks that can occur throughout all cell cycle phases. Conversely to the homologous recombination (HR) pathway, the NHEJ pathway does not need a homologous template at the DNA double-strand breaks ends and therefore NHEJ is an error-prone repair pathway that may cause mutations within the repaired region of the DNA. The NHEJ pathway is well-known to induce indels (i.e., insertions and deletions) mutations that shift the reading frame of the protein (i.e., frameshift mutations), thereby leading to a completely different translated protein from the original one.

We hope our answers were useful. Return anytime for more information and answers to any other questions you have. We hope this was helpful. Please come back whenever you need more information or answers to your queries. Thank you for visiting Westonci.ca. Stay informed by coming back for more detailed answers.