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mutations in human presenilin genes result in a higher risk for developing early onset alzheimer's disease. the human presenilin proteins from these genes alter other proteins by cleaving (cutting) them at specific positions. similar presenilin proteins were found c. elegans, a microscopic nematode worm. these nematodes do not have a brain and do not get alzheimer's disease. what function is performed by nematode presenilins?

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